Sulfated fucoidan FP08S2 inhibits lung cancer cell growth in vivo by disrupting angiogenesis via targeting VEGFR2/VEGF
Disrupted VEGF–VEGFR2 binding, reducing angiogenesis and tumor growth in A549 xenografts.
Disrupted VEGF–VEGFR2 binding, reducing angiogenesis and tumor growth in A549 xenografts.
Inhibition of lung cancer cell growth and metastasis via modulation of PI3K-Akt-mTOR pathways.
Research on the role of Smurf2-dependent ubiquitin proteasome pathway in TGFβ receptor degradation.
Fucoidan represents an ideal therapeutic agent for non-small cell lung cancer (NSCLC).
Synergy with chemotherapy for drug-resistant lung cancer cells.